AI Summary

Researchers at the Institute of Oceanology of the Chinese Academy of Sciences have identified a metabolic mechanism that enables intertidal oysters to tolerate temperature stress and linked it to malignant cell proliferation in human lung adenocarcinoma. Their study, published in PNAS, demonstrates that lung tumor cells exploit the same glycolysis‑regulating pathway to sustain energy production when faced with metabolic stress. The work highlights how a conserved marine metabolic axis can be co‑opted by cancer cells to promote growth.

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