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From DNA marks to RNA control: A new clue to acute myeloid leukemia
Acute myeloid leukemia (AML) is an aggressive blood cancer in which abnormal, immature cells build up in the bone marrow, disrupting the production of healthy blood cells. Now, a new study, led by Dr.
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A new study from the Josep Carreras Leukaemia Research Institute investigates how DNA methylation marks and RNA regulation intersect in normal blood cell development. Led by Dr. José Luis Sardina, the research focuses on TET2, a gene commonly mutated in acute myeloid leukemia that encodes a protein controlling DNA methylation, an epigenetic mechanism. By examining TET2’s role in directing genetic programs that define cell identity, the work aims to clarify how these two regulatory systems cooperate. Findings were published in Science Advances.
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