Summary

In a study led by Baylor College of Medicine and the University of Oxford, researchers found that blocking a specific molecular interaction reduced atrial fibrosis in mouse models and cells from human patients with atrial fibrillation (AF), reduced fibrotic responses in human cells, and in the mouse models reversed established atrial fibrosis and reduced abnormal heart rhythms. The study, published in the latest edition of Nature Cardiovascular Research , points to the potential for a treatment for AF that up until now has not existed.

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